Uh... English please?
"These findings revealed that C6 can be considered as an immunomodulator and that deleting C6L gene in MVA-B confers an immunological benefit by enhancing IFN-β-dependent responses and increasing the magnitude and quality of the T-cell memory immune responses to HIV-1 antigens. Our observations are relevant for the improvement of MVA vectors as HIV-1 vaccines."
That's the cliffnotes version.
"In conclusion, we show that deletion of C6L on MVA-B up-regulates IFN-β expression in human macrophages and DCs, and improves MVA-B immunogenicity in vivo, increasing the magnitude, polyfunctionality and memory T-cell responses against HIV-1 and the generation of Env-specific antibodies. Thus, VACV C6 protein interferes with host immune responses by at least in part, blocking some component(s) of the IFN-β signalling pathway. Understanding the mechanism of action of C6 will provide new insights in virus-host cell interactions and viral immunomodulation. Further work should be devoted to explore the relevance of our findings in a non-human primate model, as MVA-B ΔC6L represents a promising vector for developing HIV-1 vaccines."
Super cliffnotes version.